Thymosin Beta-4 · 10mg
Product Usage: The products we offer are intended for in-vitro laboratory research use only. The products are not for human or animal use or consumption of any kind. The products offered on this website are not intended to diagnose, cure, mitigate, treat or prevent disease. In purchasing any of these items, the customer acknowledges that there are risks involved with consumption or distribution of these products.
99.4%+ purity certificate
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How Thymosin Beta-4 works?
TB4 is widely studied as a G-actin–sequestering peptide that shifts the G-actin/F-actin balance, influencing how cells move, attach, and remodel tissue scaffolding. Mechanistic work links TB4 to angiogenic migration behaviors, and to signaling nodes including integrin-linked kinase (ILK) and inflammatory pathway regulation (e.g., NF-κB in defined cell systems). The well-known actin-binding active motif LKKTETQ is a major functional site studied in TB4 biology.
Areas of research
Area & performance
Description
Wound closure and tissue remodeling
9/10
How skin or epithelial tissue closes a defect faster (re-epithelialization, collagen organization). In rodent wound models, TB4 increased re-epithelialization versus controls and improved contraction-related healing readouts.
Angiogenesis and endothelial migration
9/10
How endothelial cells move, sprout, and form tubules during repair. TB4 is linked to endothelial migration/adhesion, tubule formation, and vessel sprouting assays.
Corneal surface repair biology
8/10
How corneal epithelium repairs after chemical injury and how inflammatory mediators shift in ocular models; TB4 accelerated corneal wound healing and reduced inflammation signals in alkali injury models.
Inflammation signaling in injury environments
8/10
How TNF-α / NF-κB-related signaling and downstream cytokine outputs change in cell models; TB4 has been shown to interfere with TNF-α-mediated NF-κB activation and IL-8 expression in mechanistic systems.
Cardiac repair hypotheses
7/10
How epicardial activation, cell survival, and migration programs behave after ischemic injury; evidence includes strong mechanistic support (ILK-linked pathways) but also mixed results depending on model timing and endpoints.
Skeletal muscle regeneration signaling
7/10
How myoblast chemotaxis and regeneration-associated programs respond after muscle injury; TB4 has been reported to increase chemotaxis in myoblast models.
Resources & citations
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Links
About peptides
Dr. Mark Hyman
MD
"Peptides represent an important element of precision anti-aging medicine."
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We make sure you are the first person to open the compound after its production. The seal means the compound is genuine, tested, and sterile.
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Compounds of other peptide companies are delivered without boxes. Best case: a bubble wrap. We developed a box that keeps the vial protected at all times.
03
Batch number
The batch number is always accessible on the packaging and the vial. You can always cross-reference it with the data base of COAs on our website for the assurance.
01
Safety seal
We make sure you are the first person to open the compound after its production. The seal means the compound is genuine, tested, and sterile.
02
Rigid packaging
Compounds of other peptide companies are delivered without boxes. Best case: a bubble wrap. We developed a box that keeps the vial protected at all times.
03
Batch number
The batch number is always accessible on the packaging and the vial. You can always cross-reference it with the data base of COAs on our website for the assurance.
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